In China, smallpox follicles were collected from the victims and placed in bottles. After a month or more, most of the smallpox viruses were dead or weak. To make someone immune, the dry wound broke in the nose. In India, pus was used from a minor case of smallpox and spots in a scratch on another person’s arm. In Turkey, Mary Wortley Montagu saw the first use of the Bovina Agency in 1717. Montagu was so impressed that he brought the idea to England. However, smallpox was sometimes strong enough to cause complete illness and eventually death. It was necessary to find a way to portray people without killing them. In 1796, Edward Jenner, an Englishman, saw that cow’s milk sometimes had smallpox, which made her immune to smallpox. Jenner continued to show that if a small dose of cowpox was deliberately administered, they would not have fatal chicken pox
What is vaccination?
Throughout history, we have developed vaccines to protect both humans and animals from a wide range of preventable and potentially dangerous diseases. With vaccines, we take advantage of one of the most important aspects of the immune system: the ability to develop immune memory. This means that once a person or animal is exposed to a particular pathogen, in other words, anything that can cause disease will develop resistance to infection with this pathogen in the future. Our adaptive immune system contains white blood cells known as T and B lymphocytes. These are activated during the first time or primary exposure of a nurse. Once the body is suffocated by the body, the T lymphocytes (known as memory cells) remain in the individual. These memory cells remain in standby mode and are ready to react quickly when a person is re-exposed to a particular probe, known as secondary exposure. Immune memory helps the immune system respond more quickly and effectively than the initial exposure. As a result, the individual is generally protected from the development of the symptoms of the disease.
Now that we know about immune memory, what about vaccines? It simply means that we are artificially generating this memory effect at an early stage to prevent future diseases. This means that we inject a weak version of pathogens, pathogens or certain parts of pathogens in the person we want to protect. It is unlikely that these pathogens cause a distinct damage, unlike their strongest powerful counterparts. In healthy individuals, these components of the vaccine activate a specific immune response that mimics the primary infection but is weak enough to cause symptoms. For example, a varicella-zoster varicella-zoster vaccine involves the injection of a small version of varicella-zoster. For healthy people, this weak virus will not cause symptoms of smallpox, but will activate the immune response. As a result, when a person encounters the varicella-zoster pollinator virus again, for example, from a person infected with chickenpox, he or she will carry a rapid immune response to the virus’s memory will not be infected with symptoms of the smallpox. By using immune memory in this way, vaccination prevents and controls the spread of a wide range of diseases, including polio, smallpox, whooping cough, measles, and seasonal influenza virus. In recent years, there has been controversy over the safety of vaccination programs. To date, all reliable scientific evidence strongly supports the importance of vaccination to prevent preventable diseases in individuals and populations.